DNP Weight Loss Steroids Powder Muscle Gain 2, 4-Dinitrophenol
|FOB Price:||US $1-6 / grams|
|Min. Order:||10 grams|
|Min. Order||FOB Price|
|10 grams||US $1-6/ grams|
|Transport Package:||Very Discreet or as Required|
|Payment Terms:||T/T, Paypal, Money Gram, Western Union|
- Model NO.: 99
- Customized: Customized
- Suitable for: Adult
- Purity: >99%
- CAS No: 51-28-5
- M.W: 184.11
- Appearance: Light Yellow Crystalline Powder
- Specification: GMP, SGS, ISO, KOSHER
- HS Code: 3001200020
- Powder: Yes
- Certification: GMP, ISO 9001, USP, BP
- State: Solid
- Product Name: 2,4-Dinitrophenol
- M.F: C6h4n2o5
- Assays: 99%
- Trademark: BETTLI
- Origin: China
Dinitrophenol is an ionophoric compound that short circuits mitochondrial ATP production. This cessation of ATP production via the mitochondria leads to an increase in the Metabolic Rate (as substrate is utilized at a higher rate so cytosolic ATP can sustain life) and subsequent weight loss.
It is one of the few chemical compounds that is causatively linked to death. Overdosing on Dinitrophenol leads to toxicity via excessive heat production, irregular breathing and heartrate, and mitochondrial dysfunction.
How to Take
Standard online protocols advice for titrating up towards a dose of 200-400 mg daily with cessation after 2 weeks of usage.
(NOTE: This dosing information is anecdotal in nature and is based on past human trials that were not conducted in a controlled fashion. No information exists in the scientific literature for the best dosages for weight loss.)
2,4-Dinitrophenol is used in the manufacture of dyes, wood preservatives, and as a pesticide. The acute (short-term) effects of 2,4-dinitrophenol in humans through oral exposure are nausea, vomiting, sweating, dizziness, headaches, and loss of weight. Chronic (long-term) oral exposure to 2,4-dinitrophenol in humans has resulted in the formation of cataracts and skin lesions, weight loss, and has caused effects on the bone marrow, central nervous system (CNS), and cardiovascular system. Limited or no information is available on the developmental, reproductive, or carcinogenic effects of 2,4-dinitrophenol in humans. EPA has not classified 2,4-dinitrophenol for carcinogenicity.
2,4-Dinitrophenol is used in the manufacture of dyes and wood preservatives, as a pesticide, and as an indicator for the detection of potassium and ammonium ions.
During the 1930s, 2,4-dinitrophenol was used as a diet pill, but this use was stopped in 1938.
2,4-Dinitrophenol (DNP) is reported to cause rapid loss of weight, but unfortunately is associated with an unacceptably high rate of significant adverse effects. DNP is sold mostly over the internet under a number of different names as a weight loss/slimming aid. It causes uncoupling of oxidative phosphorylation; the classic symptom complex associated with toxicity of phenol-based products such as DNP is a combination of hyperthermia, tachycardia, diaphoresis and tachypnoea, eventually leading to death. Fatalities related to exposure to DNP have been reported since the turn of the twentieth century. To date, there have been 62 published deaths in the medical literature attributed to DNP. In this review, we will describe the pattern and pathophysiology of DNP toxicity and summarise the previous fatalities associated with exposure to DNP.
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